Effect of Knee Hyperextension on Femoral Cartilage Thickness in Stroke Patients
Loading...
Date
2024
Authors
Journal Title
Journal ISSN
Volume Title
Publisher
Lippincott Williams & Wilkins
Open Access Color
OpenAIRE Downloads
OpenAIRE Views
Abstract
ObjectiveKnee hyperextension is one of the most common compensatory mechanisms in stroke patients. The first aim of the study was to measure knee hyperextension and femoral cartilage thickness in stroke patients. The second aim was to compare the femoral cartilage thickness of the paretic and nonparetic limbs in stroke patients with and without knee hyperextension.DesignForty stroke patients were included in the study. The patients were divided into two groups according to the presence of knee hyperextension based on kinematic analyses performed during walking with a three-dimensional motion analysis system. The medial femoral cartilage, lateral femoral cartilage, and intercondylar cartilage thicknesses of the paretic and nonparetic sides of the patients were measured by ultrasonography.ResultsIn the study group, medial femoral cartilage, intercondylar, and lateral femoral cartilage thicknesses were less on the paretic side than on the nonparetic side, while the femoral cartilage thicknesses on the paretic and nonparetic sides were similar in the control group. Paretic side medial femoral cartilage and intercondylar thicknesses were less in the study group compared with the control group, and lateral femoral cartilage thickness was similar between the two groups.ConclusionsKnee hyperextension during walking causes femoral cartilage degeneration in stroke patients.Clinical Trial code: NCT05513157ConclusionsKnee hyperextension during walking causes femoral cartilage degeneration in stroke patients.Clinical Trial code: NCT05513157
Description
Seckinogullari Korkusuz, Busra/0000-0002-8484-3659; Aritan, Serdar/0000-0001-6430-3156
Keywords
Stroke, Femoral Cartilage Thickness, Ultrasonographic Measurements, Motion Analysis
Turkish CoHE Thesis Center URL
Fields of Science
Citation
1
WoS Q
Q1
Scopus Q
Source
Volume
103
Issue
5
Start Page
371
End Page
376