Sarıçam, Ersin

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https://hdl.handle.net/20.500.14411/7015
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Ersin, Saricam
Saricam E.
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Saricam, Ersin
Sariçam E.
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Ersin, Sariçam
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Sarıçam, Ersin
Ersin, Sarıçam
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Sarıçam,E.
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Sariçam, Ersin
Saricam,E.
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ersin.saricam@atilim.edu.tr
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Internal Medical Sciences
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15840456000
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JKB-8115-2023

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International Journal of General Medicine4
Current vascular pharmacology1
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Author: Saricam, ErsinSubject: Atrial Septal Defect

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    Article
    The Evaluation of P-Wave Parameters in Patients With Percutaneous Closure of Atrial Septal Defect
    (Wiley, 2025) Astan, Ramazan; Kacmaz, Fehmi; Saricam, Ersin; Ilkay, Erdogan
    Background: Atrial septal defect (ASD) can lead to volume overload and related changes in P-wave parameters in surface electrocardiograms of these patients. In this study, we aimed to evaluate the effect of volume overload on P-wave parameters in patients with ASD. Materials and methods: This study is a retrospective cohort analysis. A total of 142 patients with secundum ASD who underwent percutaneous closure were evaluated. P-wave duration (Pmax) and P-wave dispersion (PWD) were measured on the surface ECG before and 1 h after the closure procedure. We evaluated P-wave parameters in terms of defect size, duration of the volume overload, and closure device sizes. Results: Pmax and PWD were significantly decreased after the procedure compared with the values before the procedure (p < 0.001). Pmax values had a statistically significant correlation with ASD size (< 20 mm or >= 20 mm) both before and after the procedure. Pmax values were significantly higher in patients older than 30 years of age (119.6 +/- 19.5 vs. 102.7 +/- 17.1 ms, respectively; p = 0.039). A significantly positive correlation was found between pre- and post-procedural Pmax and defect sizes (r = 0.474, p = 0.019 and r = 0.4233, p = 0.04, respectively). However, no positive correlation between PWD and defect age and size was present. Conclusion: Percutaneous closure of ASD is associated with an immediate decrease in both Pd and Pmax that seems to be related to the acute volume overload cessation in cardiac chambers.